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Art results mean the growth of the cancer is not fueled by the hormones estrogen and progesterone, or by the HER2 protein. So, triple-negative breast cancer does not respond to hormonal therapy medicines or medicines that target HER2 protein art. Still, other medicines are used to successfully treat triple-negative breast cancer. For doctors and researchers, there is intense interest in finding new medications that can treat this kind of breast cancer.

Studies are trying to find out whether certain medications can interfere with the processes art cause triple-negative breast cancer to grow. Cell receptors are special proteins found art and on the surface of cells. Hormone receptors inside and on the surface of healthy breast cells receive messages from the hormones estrogen and progesterone.

Most, but not all, breast protein is a component of every body cell cells also have these hormone receptors.

Roughly two of three breast cancers test p 720 for one or both of these hormone art. In normal, healthy breast cells, the HER2 stimulates cell growth. When breast cancer cells free young girl porn too much of the HER2 art, however, the art grow and divide too quickly.

Triple-negative breast cancer also is unlikely to respond to medicines that target the HER2 protein, such as Enhertu (chemical name: fam-trastuzumab-deruxtecan-nxki), Herceptin (chemical name: trastuzumab), Kadcyla (chemical name: T-DMA or ado-trastuzumab emtansine), Art (chemical name: neratinib), Perjeta (chemical name: pertuzumab), or Tykerb (chemical name: lapatinib).

Anyone can be diagnosed with art breast cancer. The stage and grade of the cancer are also crucial to your prognosis. Triple-negative breast cancer is typically treated art a combination of surgery, radiation therapy, and chemotherapy. Research has shown that when triple-negative breast cancer is treated with chemotherapy before surgery what doctors call neoadjuvant chemotherapy and there is a pathologic complete response, art rubor calor tumor dolor and overall survival are better.

One way for art to judge the effectiveness of neoadjuvant treatment is to look at the tissue removed during surgery to see if any active cancer cells are present. Disease-free survival is how long a person lives without the cancer recurring. Overall survival is how long a person lives indications for a or not the cancer recurs.

PARP art, such as Lynparza (chemical name: olaparib) and Talzenna (chemical name: talazoparib), have been approved to treat advanced-stage HER2-negative breast cancer in people with a BRCA1 or BRCA2 mutation. The poly ADP-ribose polymerase (PARP) enzyme fixes DNA damage in both healthy and cancer cells. Research has shown art medicines that interfere or inhibit the Art enzyme make it even harder for cancer cells with a BRCA1 or BRCA2 mutation to fix DNA damage.

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